If you reside in an apartment or house or simply want to keep your home free from EMFs There are a variety of methods to reduce exposure. One of the easiest is to limit the use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your house. Another method to protect your home against EMF radiation is to use a RF shielding canopy. This is a cloth made of net which contains EMF shielding and is used to prevent EMFs from entering a room. Another option is to have your house equipped with a conductive enclosure. These devices are called Faraday cages.
Several studies have shown studies have shown that nonionizing EMF can cause antiproliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to involve down-regulation of cancer stem cells. This may account for the long-term responses seen in some patients with advanced HCC. But, emf blockers for AM EMF's effects on patients with cancer is not clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. One group did not have exposure to RF EMF. Second group members were exposed RF EMF at a frequency that is similar to the frequency used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism of tumor-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. emf blocker on HCC cells is caused by CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications for treatment and diagnosis of many cancers.
The tumours in the control group were not exposed EMF from RF, and fed a standard mouse diet. The tumours in those in the HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumors were removed after they had a high burden.
The tumours from the three groups showed different growth curves. The tumors treated with HCCMF saw a significant reduction in size of the tumor after 8 weeks. However, the tumours which were treated by RCF showed no signs of shrinkage. The difference was highly significant. The tumors treated with RCF were able to show necrosis, which is common in tumours that are exposed to RCF. It is possible that the necrosis was due to an absence of oxygen in larger cancers.
In conclusion, the findings suggest the fact that AM EMF has anticancer effects in vitro and in live. emf blockers of studies have proven that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF produces these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained effect on the development of HCC tumours in living tissue.