Additional info if you live in an apartment or house or simply need to ensure that your house is free of electromagnetic fields There are plenty options to limit your exposure. One of the easiest is to restrict your electronic device use. emf blockers is also possible to use EMF blocker paint to block EMF radiation from entering your house. Another way to shield your house from EMF radiations is to use a RF shielding canopy. This is a cloth of net that contains EMF shielding and is used to block EMFs from entering rooms. Another alternative is to have your house equipped with an electrical enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated studies have shown that nonionizing EMF produces antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to involve down-regulation in cancer-related stem cells. This may account for the long-term response seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients suffering from cancer isn't clear.
Aspects from AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage in the tumour.
The reason for tumour-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is controlled through CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications for the treatment and diagnosis of many cancers.
The tumours of the controls were never exposed EMF from RF, and fed a normal diet of mice. The tumors in the HCCMF group were treated with Huh7 cells when they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in size of the tumor after eight weeks. However, the tumors which were treated by RCF did not show any shrinkage. The difference was highly significant. emf blockers treated with RCF were able to show necrosis, which is typical in tumors that have been exposed to RCF. It is possible that the necrosis was due to a lack of oxygen in the larger tumours.
In conclusion, the findings indicate the fact that AM EMF exhibits anticancer properties in vitro as well as in vivo. A number of studies have proven that AM RF EMF produces measurable tumour shrinkage in HCC patients. It is possible that AM RF EMF causes these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in vivo.
