No matter if you live in a house or apartment or simply want to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from entering your house. Another way to shield your home against EMF radiation would be to use an RF shielding canopy. It is a type of net that contains EMF shielding and is used to block EMFs from entering a space. Another alternative is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
A number of studies have proven how the non-ionizing energy of RF has antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed result from the deregulation of cancer stem cells. This may account for the long-term response seen in some patients with advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not yet clear.
Aspects on the effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumours were divided in three different groups. One group did not have exposure RF EMF. Second group members were exposed to RF EMF at frequencies similar to the frequency used by humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumors was assessed against the effect of RCF. The results showed that the tumors treated by HCCMF showed significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism of tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled through CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H may have broader implications for the diagnosis and treatment of various cancers.
emf blocker in those in the group that were unaffected to EMF from radiofrequency, and fed a normal diet of mice. The tumors of those in the HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.
The tumors of the three groups showed different growth curves. The tumours treated with HCCMF saw a significant reduction in the size of the tumour after 8 weeks. However, emf blocking that were treated using RCF showed no reduction in size. The difference was substantial. The tumors treated with RCF showed necrosis, which is common in tumors that have been that are exposed to RCF. It is possible that this necrosis was due to a lack of oxygen in the more invasive cancers.
In sum, the results show an AM-RF EMF exhibits anticancer effects in vitro and in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In emf blocking , AM RF EMF may cause a lasting influence on the growth of HCC tumours in vivo.